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Cellular uptake of folate-olaparib conjugates via folate receptor-mediated endocytosis: Potential for selective delivery of DNA damage response inhibitors into tumour cells

V. Bavetsias, L. Pickard, S. Diaz-Sanchez, S. Gowan, F. Wood, R. Ruddle, C. Wong, H. Sharif, F. Raynaud, R. Burke, A. Wang, J. Blagg, V. Kirkin, O. Rossanese, U. Banerji

PreprintUso en el mundo real

En palabras de los autores

The folate receptor (FR) is overexpressed in a range of human tumours including ovarian cancer cells. We propose that the overexpression of the FR on the surface of ovarian tumour cells could be exploited for the selective delivery of a DNA damage response inhibitor (DDRi) in the form of an intact folate drug conjugate (FDC). This approach would improve the therapeutic index of the parent DDRi facilitating combination studies of the DDRi-based FDC with DNA damaging chemotherapy. FR-mediated cellular uptake of the proposed folate drug conjugates is requisite for FDC selective delivery into tumours. In this study, we synthesised a series of olaparib-based folate conjugates that maintained the biochemical PARP1 inhibition associated with olaparib and showed binding affinity for the folate receptor. Significantly, we identified compounds 10b and 11 that selectively enter FR overexpressing tumour cells via folate receptor-mediated endocytosis in their intact form and engage with their target as demonstrated by the potent inhibition of PARylation (KB cells, PARylation IC50 = 5.7 and 3.9 nM; respectively).

Resultado principalEl resumen no menciona limitaciones.

Apareció: viernes, 25 de septiembre. bioRxiv. Preprint, todavía sin revisión por pares.

DOI: 10.64898/2026.09.23.752340