Direct cellular contact by Porphyromonas gingivalis suppresses nociceptor excitability and induces analgesia during inflammation
In the authors' words
This journal does not let us republish the full abstract. Here are the two sentences Pipette selected, quoted from it. Read the rest at the publisher.
Main result
These findings define a contact-dependent mechanism that actively suppresses peripheral pain signaling during inflammation.
Appeared: Sunday, September 27. Science Advances. Peer-reviewed journal.